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find Author "黄妹" 8 results
  • 伴胸水的结节病一例报告

    Release date:2016-08-30 11:56 Export PDF Favorites Scan
  • 24例肺淋巴管癌病临床分析并文献回顾

    肺淋巴管癌病(PLC)是一种特殊形式的肺内转移癌,以转移性癌细胞在淋巴管内弥漫性生长、形成癌栓为特征。常见于腺癌、鳞癌及神经内分泌性肿瘤的肺内转移。胸部CT表现为小叶间隔增厚,形成网状、 网结节状改变,或支气管血管束显著增粗,酷似肺间质病变,因此容易误诊。现将我院2005年8月至2011 年4月收治的24例PLC病例分析如下,以强化临床医生对本病的重视,减少误诊、漏诊的发生。临床资料 24例患者中,男8例,女16例,男女比例为1:2;年龄37~58岁。确诊方法包括经支气管肺活检术(TBLB)16例次,痰脱落细胞学阳性4例次,颈部淋巴结穿刺术6例次,胸水脱落细胞学阳性1例次。24例患者均有咳嗽症状,为刺激性干咳。气喘20例,胸闷4例,痰中带血2例;体格检查发现湿啰音4例,干啰音2例,爆裂音2例,呼吸音减低1例,大多数患者无明显的肺部体征。肺功能显示5例患者为轻到中度限制性通气功能障碍,弥散功能减退,19例患者未行肺功能检查。胸部高分辨CT(HRCT)均表现为不同程度的小叶间隔不均匀性增厚及支气管血管束结节状增粗,伴或不伴有胸膜增厚、胸水形成及纵隔淋巴结肿大等。24例患者胸部CT表现双侧均有病变,其中9例患者以单侧病变为主。8例为原发肺癌伴肺内淋巴管转移,15例为肺外转移癌(乳腺癌5例,胃癌4例,肝癌1例,食管癌1例,宫颈癌1例,胰腺癌1例,甲状腺癌1例,结肠癌1例)伴肺内淋巴管转移,1例至患者出院为止未能找到原发部位。院外误诊情况:12例误诊为肺间质病变,给予激素治疗无效;8例误诊为肺部感染或肺炎,2例误诊为支气管炎,抗感染治疗无效;2例误诊为结核性胸膜炎,给予抗结核治疗无效。

    Release date:2016-08-30 11:58 Export PDF Favorites Scan
  • Sweet 综合征伴机化性肺炎一例报告及文献复习

    目的 提高临床医生对Sweet 综合征合并血液病、肺部疾病的认识, 探讨血液病合并 Sweet综合征的临床特征。方法 报告1 例Sweet 综合征并机化性肺炎、骨髓增生异常综合征的病例, 并作文献复习。结果 血液病合并Sweet 综合征是一种全身性、多系统性疾病, 除皮损外尚可侵犯其他器官, 如肺脏、肝脏、脾脏、肌肉、关节等。结论 报告1 例经活检由病理诊断的Sweet 综合征同时侵犯肺脏引起机化性肺炎的病例, 在糖皮质激素治疗皮损的同时应积极治疗血液病。

    Release date:2016-09-13 04:07 Export PDF Favorites Scan
  • 吸烟相关间质性肺疾病的对比分析

    目的提高对不同类型的吸烟相关间质性肺疾病的临床、影像及病理学特点的认识。方法对诊断为呼吸性细支气管炎伴间质性肺疾病(RB-ILD)、脱屑性间质性肺炎(DIP)、肺朗格汉斯组织细胞增生症(PLCH)患者的临床表现、影像学和组织病理学特点进行分析比较。结果3 例患者,男 2 例,女 1 例,年龄 21~71 岁,吸烟史为 4~50 年,均为慢性起病,主要临床表现为咳嗽、咳痰、活动后气促。胸部高分辨 CT 的主要表现为小叶中央型微结节影、网格影、囊状影。病理学表现:RB-ILD 呼吸性细支气管周围少许炎症渗出、管腔中有少许巨噬细胞;DIP 肺泡腔内巨噬细胞聚集;PLCH 以朗格汉斯细胞组成为主的星状结节,朗格汉斯组织细胞表达 CD1α。结论吸烟相关的间质性肺疾病临床及影像表现各异,增强对这类疾病的认识,结合组织病理学检查有利于及早诊断这类疾病。

    Release date:2020-11-24 05:41 Export PDF Favorites Scan
  • 一例表现为双肺弥漫囊状改变的原发性肺腺癌的影像学演变过程

    Release date:2021-06-30 03:37 Export PDF Favorites Scan
  • The Value of Serum KL-6 Level as An Diagnostic Indicator in Patients with Interstitial Lung Diseases

    ObjectiveTo determine the diagnostic value of serum KL-6 level in patients with interstitial lung diseases (ILD). MethodsAll the ILD patients enrolled were hospitalized from April 2013 to April 2014. Patients with other pulmonary diseases and healthy subjects were chosen as control groups simultaneously. Serum KL-6 concentrations were measured by chemiluminescent enzyme immunoassay. The association with serum KL-6 level and pulmonary function was analyzed. ResultsThere were 149 ILD patients, 155 patients with other pulmonary diseases, and 64 healthy subjects. The average serum levels of KL-6 were (1 801.86±2 831.36) U/mL, (267.00±124.41) U/mL, (201.28±81.18) U/mL in the patients with ILD, the patients with other pulmonary diseases and the healthy controls, respectively. The sensitivity and the specificity of the serum KL-6 for the diagnosis of ILD was 83.89% and 92.24% respectively when the cut-off level was set at 500 U/mL. The Kappa value was 0.767 (P < 0.001). The best cut-off value of KL-6 was 469.5 U/mL. Serum KL-6 levels in the patients with ILD were significantly higher compared with the patients with chronic obstructive pulmonary disease, pneumonia, tuberculosis, bronchiectasis and the healthy controls, respectively (all P < 0.001). The KL-6 levels in the pulmonary alveolar proteinosis patients were significantly higher compared with the patients with cryptogenic organizing pneumonia (COP), the patients with idiopathic pulmonary fibrosis (IPF) and the patients with connective tissue disease (CTD-ILD) (all P < 0.001). While the KL-6 concentration in IPF and CTD-ILD were significantly higher than that in COP (P=0.003 and P=0.008, respectively). Significant negative correlations were found between the levels of serum KL-6 and vital capacity as a percentage of the predicted value, forced vital capacity as a percentage of the predicted value, forced expiratory volume in one second as a percentage of the predicted value and carbon monoxide diffusing capacity as a percentage of the predicted value (all P < 0.001). Follow-up study showed the levels of serum KL-6 were consistent with clinical efficacy. ConclusionSerum KL-6 level is a reliable serum marker for ILD, and is related with the severity of disease and clinical efficacy.

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  • Analysis on the Clinical Characteristics and Survival of 53 Patients with Pulmonary Fibrosis Complicated with Lung Cancer

    ObjectiveTo analyze the clinical characteristics and survival of pulmonary fibrosis (PF) patients complicated with lung cancer (LC) (PL-LC). MethodsFifty-three patients with PF diagnosed as LC from January 2008 to March 2014 in Nanjing Drum Tower Hospital were included in this study. Univariate analysis and Cox regression analysis were used to detect the effects of clinical variables on survival. Kaplan-Meier method was used to calculate the median survival time (MST) and overall survival (OS). ResultsMale patients (n=48, 90.6%) and patients with a history of smoking (n=42, 79.2%) were more easily suffered from PF-LC. The average age was 68.6±9.5 years. Cox multivariate analysis revealed that Velcro crackles (P=0.009) and clinical stage (P=0.013) were the independent risk factors of survival in the patients with PF-LC. The MST of 53 patients was 6.0 months.The survival rates of 1-year and 2-year were 34.1% and 22.0%, respectively. Forty-two (79.2%) patients were idiopathic pulmonary fibrosis (IPF) complicated with LC, and 11 (20.8%) patients were secondary pulmonary fibrosis (SPF) complicated with LC. OS difference between two groups was not significant (P=0.610). OS of NSCLC group (n=37) was significantly prolonged than that of SCLC group (n=6) and unclassified pathological pattern group (n=10) (P=0.035). OS of Ⅰ and Ⅱstage patients (n=13) was significantly longer than that of Ⅲ and Ⅳ stage patients (n=40) (P=0.002). MST and OS of patients with LC treated (n=31) were significantly better than those of untreated patients (n=22) (P < 0.001) and OS of patients treated by comprehensive therapy (n=11) was significantly prolonged than that of patients treated by mono-therapy (n=20) (P=0.036). ConclusionsVelcro crackles and clinical stages are the independent risk factors of prognosis in PF-LC patients. It is beneficial to survival if the PF patients with LC were treated by comprehensive therapy.

    Release date:2016-10-10 10:33 Export PDF Favorites Scan
  • Pulmonary lymphangitic carcinomatosis from gastric signet ring cell carcinoma: three cases report and literature review

    ObjectiveTo investigate the clinical, radiological and pathological features of pulmonary lymphangitic carcinomatosis (PLC) from gastric signet ring cell carcinoma.MethodsThe data of 3 cases of PLC from gastric signet ring cell carcinoma were analyzed and relevant literature was reviewed. The electronic databases of Wanfang, VIP, CNKI and PubMed were searched by using the keywords " pulmonary lymphangitic carcinomatosis” and " gastric signet ring cell carcinoma”.ResultsThe average age of three patients was 34 years (ranging from 20 to 50 years). There were 2 males and 1 female. The three patients presented with non-specific respiratory complaints of cough and shortness of breath. Thickening of interlobular septa was the major radiographical abnormality. Mediastinal lymphadenopathy was seen in 1 patient. Bilateral pleural effusion was seen in 2 patients. However, gastroscopy revealed metastatic gastric signet ring cell carcinoma with the presence of lymphagitic carcinomatosis. One patient survived more than seven months with systemic chemotherapy began on day 7 of hospitalization based on the follow-up information available. Literature review found 2 cases of PLC from gastric signet ring cell carcinoma. Two cases were female aged 45 and 59 years. The chief complaints were dry cough and shortness of breath. The survival time was less than 1 month from diagnosis.ConclusionsPLC from gastric signet ring cell carcinoma is easily misdiagnosed as primary pulmonary diseases. Early identification, diagnosis and treatment are crucial to improve the survival of PLC patients.

    Release date:2017-07-24 01:54 Export PDF Favorites Scan
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