Progress in Pathogenesis of Cholelithiasis_CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY

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别平 , 范毓东

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胆囊结石; 发病机理; 致石基因

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Citation： 别平,范毓东. Progress in Pathogenesis of Cholelithiasis. CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY, 2003, 10(5): 439-441. doi: Copy

1.	Moraga ML, Rocco P, Miquel JF, et al. Mitochondrial DNA polymorphisms in Chilean aboriginal populations: implications for the peopling of the southern cone of the continent [J]. Am J Phys Anthropol, 2000； 113(1)∶19.
2.	Wang DQ,Paigen B,Carey MC. Phenotypic characterization of Lithgenes that determine susceptibility to cholesterol cholelithiasis in inbred mice: physicalchemistry of gallbladder bile [J]. J Lipid Res, 1997; 38(7)∶1395.
3.	Paigen B，Schork NJ，Svenson KL，et al. Quantitative trait loci mapping for cholesterol gallstones in AKR/J and C57L/J strains of mice [J]. Physiol Genomics, 2000； 4(1)∶59.
4.	Lammert F, Wang DQ, Wittenburg H, et al. Lith genes control mucin accumulation, cholesterol crystallization, and gallstone formation in A/J and AKR/J inbred mice [J]. Hepatology, 2002； 36(5)∶1145.
5.	Wittenburg H, Lammert F, Wang DQ, et al. Interacting QTLs for cholesterol gallstones and gallbladder mucin in AKR and SWR strains of mice [J]. Physiol Genomics, 2002； 8(1)∶ 67.
6.	Mittal B, Mittal RD. Genetics of gallstone disease [J]. J Postgrad Med, 2002； 48(2)∶149.
7.	Shoda J, He BF, Tanaka N, et al. Primary dual defect of cholesterol and bile acid metabolism in liver of patients with intrahepatic calculi [J]. Gastroenterology, 1995； 108(5)∶1534.
8.	Kozarsky KF, Donahee MH, Rigotti A, et al. Overexpression of the HDL receptor SRBI alters plasma HDL and bile cholesterol levels [J]. Nature, 1997； 387(6631)∶414.
9.	Russell DW. Nuclear orphan receptors control cholesterol catabolism [J]. Cell, 1999； 97(5)∶539.
10.	Cali JJ, Russell DW. Characterization of human sterol 27hydroxylase. A mitochondrial cytochrome P450 that catalyzes multiple oxidation reaction in bile acid biosynthesis [J]. J Biol Chem, 1991； 266(12)∶7774.
11.	Miyasaka K, Takata Y, Funakoshi A. Association of cholecystokinin A receptor gene polymorphism with cholelithiasis and the molecular mechanisms of this polymorphism [J]. J Gastroenterol, 2002； 37(Suppl 14)∶102.
12.	Lu W, Tang D, Cao S, et al. The study on the relationship between the expression of calponin and gallstone formation [J]. J Tongji Med Univ, 1997； 17(2)∶86.
13.	Swidsinski A, Lee SP. The role of bacteria in gallstone pathogenesis [J]. Front Biosci, 2001； 6∶E93.
14.	Kawai M, Iwahashi M, Uchiyama K, et al. Grampositive cocci are associated with the formation of completely pure cholesterol stones [J]. Am J Gastroenterol, 2002； 97(1)∶83.
15.	孙立江, 刘刚, 程力, 等. 雌性激素与胆石成因的实验及临床研究 [J]. 肝胆胰外科杂志, 2000； 12(1)∶37.
16.	Ahrendt SA, McGuire GE, Pitt HA, et al. Why does somatostatin cause gallstones? [J]. Am J Surg, 1991； 161(1)∶177.
17.	Conter RL, Roslyn JJ, PorterFink V, et al. Gallbladder absorption increases during early cholesterol gallstone formation [J]. Am J Surg, 1986； 151(1)∶184.
18.	Afdhal NH, Niu N, Gantz D, et al. Bovine gallbladder mucin accelerates cholesterol monohydrate crystal growth in model bile [J]. Gastroenterology, 1993； 104(5)∶1515.

1. Moraga ML, Rocco P, Miquel JF, et al. Mitochondrial DNA polymorphisms in Chilean aboriginal populations: implications for the peopling of the southern cone of the continent [J]. Am J Phys Anthropol, 2000； 113(1)∶19.
2. Wang DQ,Paigen B,Carey MC. Phenotypic characterization of Lithgenes that determine susceptibility to cholesterol cholelithiasis in inbred mice: physicalchemistry of gallbladder bile [J]. J Lipid Res, 1997; 38(7)∶1395.
3. Paigen B，Schork NJ，Svenson KL，et al. Quantitative trait loci mapping for cholesterol gallstones in AKR/J and C57L/J strains of mice [J]. Physiol Genomics, 2000； 4(1)∶59.
4. Lammert F, Wang DQ, Wittenburg H, et al. Lith genes control mucin accumulation, cholesterol crystallization, and gallstone formation in A/J and AKR/J inbred mice [J]. Hepatology, 2002； 36(5)∶1145.
5. Wittenburg H, Lammert F, Wang DQ, et al. Interacting QTLs for cholesterol gallstones and gallbladder mucin in AKR and SWR strains of mice [J]. Physiol Genomics, 2002； 8(1)∶ 67.
6. Mittal B, Mittal RD. Genetics of gallstone disease [J]. J Postgrad Med, 2002； 48(2)∶149.
7. Shoda J, He BF, Tanaka N, et al. Primary dual defect of cholesterol and bile acid metabolism in liver of patients with intrahepatic calculi [J]. Gastroenterology, 1995； 108(5)∶1534.
8. Kozarsky KF, Donahee MH, Rigotti A, et al. Overexpression of the HDL receptor SRBI alters plasma HDL and bile cholesterol levels [J]. Nature, 1997； 387(6631)∶414.
9. Russell DW. Nuclear orphan receptors control cholesterol catabolism [J]. Cell, 1999； 97(5)∶539.
10. Cali JJ, Russell DW. Characterization of human sterol 27hydroxylase. A mitochondrial cytochrome P450 that catalyzes multiple oxidation reaction in bile acid biosynthesis [J]. J Biol Chem, 1991； 266(12)∶7774.
11. Miyasaka K, Takata Y, Funakoshi A. Association of cholecystokinin A receptor gene polymorphism with cholelithiasis and the molecular mechanisms of this polymorphism [J]. J Gastroenterol, 2002； 37(Suppl 14)∶102.
12. Lu W, Tang D, Cao S, et al. The study on the relationship between the expression of calponin and gallstone formation [J]. J Tongji Med Univ, 1997； 17(2)∶86.
13. Swidsinski A, Lee SP. The role of bacteria in gallstone pathogenesis [J]. Front Biosci, 2001； 6∶E93.
14. Kawai M, Iwahashi M, Uchiyama K, et al. Grampositive cocci are associated with the formation of completely pure cholesterol stones [J]. Am J Gastroenterol, 2002； 97(1)∶83.
15. 孙立江, 刘刚, 程力, 等. 雌性激素与胆石成因的实验及临床研究 [J]. 肝胆胰外科杂志, 2000； 12(1)∶37.
16. Ahrendt SA, McGuire GE, Pitt HA, et al. Why does somatostatin cause gallstones? [J]. Am J Surg, 1991； 161(1)∶177.
17. Conter RL, Roslyn JJ, PorterFink V, et al. Gallbladder absorption increases during early cholesterol gallstone formation [J]. Am J Surg, 1986； 151(1)∶184.
18. Afdhal NH, Niu N, Gantz D, et al. Bovine gallbladder mucin accelerates cholesterol monohydrate crystal growth in model bile [J]. Gastroenterology, 1993； 104(5)∶1515.

CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY

Progress in Pathogenesis of Cholelithiasis

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