1. |
Carey LA, Perou CM, Livasy CA, et al. Race, breast cancer subtypes, and survival in the Carolina Breast Cancer Study[J]. JAMA, 2006, 295(21):2492-2502.
|
2. |
Patel NS, Li JL, Generali D. et al. Up-regulation of delta-like 4 ligand in human tumor vasculature and the role of basal expression in endothelial cell function[J]. Caner Res, 2005, 65(19):8690-8697.
|
3. |
Wang YY, Ye ZY, Zhao ZS, et al. High-level expression of S100A4 correlates with lymph node metastasis and poor prognosis in patients with gastric cancer[J]. Ann Surg Oncol, 2010, 17(1): 89-97.
|
4. |
Boye K, Maelandsmo GM. S100A4 and metastasis: a small actor playing many roles[J]. Am J pathol, 2010, 176(2):528-535.
|
5. |
Singletary SE, Allred C, Ashley P, et al. Staging system for breast cancer:revisions for the 6th edition of the AJCC Cancer Staging Manual[J]. Surg Clin North Am, 2003, 83(4):803-819.
|
6. |
Kopfstein L, Christofori G. Metastasis:cell-autonomous mechanisms versus contributions by the tumor microenviroment[J]. Cell Mol Life Sci, 2006,63(4): 449-468.
|
7. |
Bergers G, Benjamin LE. Tumorigenesis and the angiogenic switch[J]. Nat Rev Cancer, 2003, 3(6):401-410.
|
8. |
Joyce JA. Therapeutic targeting of the tumor microenvironment[J]. Cancer Cell, 2005, 7(6):513-520.
|
9. |
Kerbel RS. Tumor angiogenesis[J]. N Engl J Med, 2008, 358(19):2039-2049.
|
10. |
Thurston G, Noguera-Troise I, Yancopoulos GD. The delta paradox: DLL4 blockade leads to more tumour vessels but less tumour growth[J]. Nat Rev Cancer, 2007, 7(5):327-331.
|
11. |
Bray SJ. Notch signaling:a simple pathway becomes complex[J]. Nat Rev Mol Cell Biol, 2006, 7(9):678-689.
|
12. |
Benedito R, Trindade A, Hirashima M, et al. Loss of Notch signalling induced by DLL4 causes arterial calibre reduction by increasing endothelial cell response to angiogenic stimuli[J]. BMC Dev Biol, 2008, 8:117.
|
13. |
Li JL, Sainson RC, Shi W, et al. Delta-like 4 Notch ligand regulates tumor angiogenesis, improves tumor vascular function, and promotes tumor growth in vivo[J]. Cancer Res, 2007, 67 (23): 11244-11253.
|
14. |
Buchler P, Gazdhar A,Schubert M, et al. The Notch signaling pathway is related to neurovaseular progression of pancreaticcancer[J]. Ann Surg, 2005, 242(6):791-801.
|
15. |
Mullendore ME, Koorstra JB, Li YM, et al. Ligand-dependent Notch signaling is involved in tumor initiation and tumor maintenance in pancreatic cancer[J]. Clin Cancer Res, 2009, 15(7):2291-2301.
|
16. |
Martinez JC, Muller MM,Turley H, et al. Nuclear and membrane expression of the angiogenesis regulator delta-like ligand 4 (DLL4) in normal and malignant human tissues[J]. Histopathology, 2009, 54(5):598-606.
|
17. |
Grigorian M, Lukanidin E. Activator of metastasis in cancer cells, Mst1/S100A4 protein binds to tumor suppressor protein p53[J]. Genetika, 2003, 39(7): 900-908.
|
18. |
Fernandez-Fernandez MR, Veprintsev DB, Fersht AR. Proteins of the s100 family regulate the oligomerization of p53 tumorsuppressor[J]. Proc Natl Acad Sci USA, 2005, 102(13):4735-4740.
|
19. |
Lee WY, Su WC, Lin PW, et al. Expression of S100A4 and Met: potential predictors for metastasis and survival in early-stage breast cancer[J]. Oncology, 2004, 66(6):429-438.
|
20. |
李欣, 张丽杰, 宋英. S100A4蛋白在乳腺癌组织的表达及临床意义[J]. 现代肿瘤医学, 2008,16(3):359-361.
|
21. |
Rudland PS, Platt Higgins A, Renshaw C, et al. Prognostic significance of the metastasis-inducing protein S100A4 (p9Ka) in human breast cancer[J]. Cancer Res, 2000, 60(6): 1595-1603.
|
22. |
Ambartsumian N, Grigorian M, Lukanidin E. Genetically modified mouse models to study the role of metastasis-promoting S100A4 (mts1) protein in metastatic mammary cancer[J].J Dairy Res, 2005, 72 (S1): 27-33.
|
23. |
Schmidt-Hansen B, Ornas D, Grigorian M, et al. Extracellular S100A4 (mts1) stimulates invasive growth of mouse endothelial cells and modulates MMP-13 matrix metalloproteinase activity[J]. Oncogene, 2004, 23(32): 5487-5495.
|
24. |
鲁林源, 艾开兴, 黄新余, 等. S100A4、VEGF的表达与胰腺癌预后的关系[J]. 肿瘤, 2008, 28(4): 326-329.
|
25. |
Semov A, Moreno MJ, Onichtchenko A, et al. Metastasis-associated protein S100A4 induces angiogenesis through interaction with AnnexinⅡand accelerated plasmin formation[J]. J Biol Chem, 2005, 280(21): 20833-20841.
|